Use of tgf-.beta. antagonists to treat or to prevent chronic...

A - Human Necessities – 61 – K

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A61K 31/00 (2006.01) A61K 31/194 (2006.01) A61K 31/70 (2006.01) A61K 31/7004 (2006.01) A61K 38/00 (2006.01) A61K 38/17 (2006.01) A61K 38/18 (2006.01) A61K 38/22 (2006.01) A61K 38/30 (2006.01) A61K 38/31 (2006.01) C07K 14/495 (2006.01) A61K 48/00 (2006.01)

Patent

CA 2473829

Effective use of a TGF-.beta. antagonist to treat or to prevent loss of transplant function is described herein. Use of a TGF-.beta. antagonist is demonstrated to effectively prevent loss of organ function in a host due to chronic rejection in which TGF-.beta.-mediated fibroproliferation is a characteristic. Expression in situ of a TGF-.beta. antagonist in the form of a recombinant receptor, i.e., TGF-.beta. type III receptor (TGFBIIIR) showed prevention of bronchiolitis obliterans in comparison to untreated controls in a rat lung transplant model. This provides an effective method for preventing or inhibiting chronic rejection of transplant organs such as lung, kidney, liver and hear in vertebrate hosts including human hosts.

L'invention concerne l'utilisation efficace d'un antagoniste de TGF-.beta. dans le traitement ou la prévention de la perte des fonctions du transplant. L'utilisation d'un antagoniste de TGF-.beta. s'est avérée efficace dans la prévention de la perte de fonction d'un organe chez un hôte en raison du rejet chronique, caractérisé par la fibroprolifération à médiation par TGF-.beta.. L'expression in situ d'un antagoniste TGF-.beta. sous la forme d'un récepteur recombinant, c'est-à-dire d'un récepteur de TGF-.beta. de type III (TGFBIIIR) a permis la prévention de bronchiolitis obliterans en comparaison aux modèles de contrôle dans un modèle de transplantation du poumon de rat. L'invention propose un procédé efficace de prévention ou d'inhibition du rejet chronique d'organes transplantés tels que poumon, rein, foie et oreille chez les hôtes vertébrés, y compris les humains.

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