Trpv1+ sensory neurons control of beta-cell stress and islet...

A - Human Necessities – 61 – K

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A61K 38/08 (2006.01) A61K 38/02 (2006.01) A61P 3/10 (2006.01)

Patent

CA 2672490

A process is disclosed for controlling inflammatory tissue access through release of neuropeptides, such as substance P (sP), to insulin-responsive sensory neurons, whereby simultaneous control of insulin sensitivity/resistance is manifested. In models of Type 1 and Type 2 diabetes, sensory afferents, in particular TRPV1, have fundamental roles in insulin/glucose homeostasis, islet physiology and autoimmune tissue inflammation. By manipulation of the TRPV1 neuro-.beta.-cell circuit or enhancement of pancreatic sP levels, normalization of insulin resistance, clearance of inflammation and prevention of both Type 1 and Type 2 diabetes is realized.

La présente invention concerne un procédé destiné à contrôler l'accès aux tissus inflammatoires par la libération de neuropeptides, telle que la substance P (sP), des neurones sensoriels sensibles à l'insuline, moyennant quoi le contrôle simultané de la sensibilité/résistance à l'insuline est démontré. Dans des modèles des diabètes de type 1 et de type 2, des afférents sensoriels, en particulier le TRPV1, ont des rôles fondamentaux dans l'homéostasie de l'insuline/glucose, la physiologie des îlots et l'inflammation auto-immune des tissus. La normalisation de la résistance à l'insuline, la fin de l'inflammation et la prévention à la fois des diabètes de type 1 et de type 2 sont obtenues par la manipulation du circuit des .beta.-cellules neurologiques TRPV1 ou de l'augmentation des niveaux de la sP pancréatique.

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